维普期刊资源整合服务平台（V6.5）

30篇 您的检索式：作者名="Knoblach S"

选中清除导出查看参考文献查看引证文献引用追踪共2页首页上一页第1页下一页末页 /2 跳转

	题名	作者	年代	出处	被引量
1	Dystrophin-deficient cardiomyopathy in mouse: expression of Nox4 and Lox are associated with fibrosis and altered functional parameters in the heart 显示文摘	SPURNEY CF KNOBLACH S PISTILLI EE	2008	Neuromuscul Disord2008，18，5：	1
2	Gene profiling in spinal cord injury shows role of cell cycle in neuronal death显示文摘	Di Giovanni S Knoblach S M Brandoli C	2003	Annals of neurology2003，53，4：	1
3	Gene profiling in spinal cord injury shows role of cell cycle in neuronal death显示文摘	di GIOVANNI S KNOBLACH S M BRANDOL C	2003	Annals of neurology2003，53，4：	1
4	Activa- tion of CPP32 like Caspase contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury显示文摘	YAKOVLEV A G KNOBLACH S M FAN I	1997	J Neurosci1997，17，：	1
5	Diffusion and high resolution MRI of traumatic brain injury in rats: time course and correlation with histology显示文摘	Albensi B C Knoblach S M Chew B G	2000	Exp Neurol2000，162，1：	1
6	Multiple caspases are activated after traumatic brain injury:evidence for involvement in functional outcome显示文摘	Knoblach S M Nikolaeva M Huang X	2002	J Neurotrauma2002，19，10：	1
7	Multiple caspases are activated after traumatic brain injury:evidence for involvement in functional outcome 显示文摘	KNOBLACH S M NIKOLAEVA M Huang K	2002	J Neurotrauna2002，19，：	1
8	Multiple caspases are activated after traumatic brain injury:evidence for involvement in functional outcome显示文摘	Knoblach S M Nikolaeva M Huang X	2002	J Neurotrauma2002，19，10：	1
9	Inerleukin-10 improves out- come and alters proinflammatory cytokine expression after experimental traumatic brain injury显示文摘	Knoblach S M Faden A I	1998	Exp Neurol1998，153，：	1
10	Activation of CPP32-like caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury显示文摘	Yakovlev A G Knoblach S M Fan L	1997	J Neurosci1997，17，19：	1
11	Activation of CPP32-like Caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury显示文摘	Yakovlev A G Knoblach S M Fan I	1997	J Neurosci1997，17，：	1
12	Activation of CPP32-Like Caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury显示文摘	YAKOVLEV A G KNOBLACH S M FAN L	1995	Int J Cancer1995，70，：	1
13	Multiple caspases are activated after traumatic brain injury:evidence for involvement in functional outcome显示文摘	Knoblach S M Nikolaeva I M Huang X	2002	J Neurotrauma2002，19，10：	1
14	Diversity of the pro- tein disulfide isomerase family: identification of breast tumor in- duced Hag2 and Hag3 as novel members of the protein family 显示文摘	Persson S Rosenquist M Knoblach B	2005	Mol Phylogenet Evol2005，36，3：	1
15	New in vitro model of traumatic neuronal injury: evaluation of secondary injury and glutamate receptor-mediated neurotoxicity 显示文摘	MUKHIN A G IVANOVA S A KNOBLACH S M	1997	J Neurotrauma1997，14，9：	1
16	Gene profil- ing in spinal cord injury shows role of cell cycle in neuronal death 显示文摘	Di Giovanni S Knoblach SM Brandoli C	2003	Ann Neurol2003，53，4：	1
17	Activation of CPP32line caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury显示文摘	Yakovlev A G Knoblach S M Fan I	1997	J Neurosci1997，17，：	1
18	New in vitro model of traumatic neuronal injury: evaluation of secondary injury and glutamate receptor-mediated neurotoxicity 显示文摘	Muhkin A G Ivanova S A Knoblach S M	1997	J Neurotrauma1997，14，9：	1
19	Diversity of the Protein Disulfide Isomerase Family: Identification of Breast Tumor Induced Hag2 and Hag3 as Novel Members of the Protein Family显示文摘	PERSSON S ROSENQUIST M KNOBLACH B	2005	Mol Phy- logenet Evol2005，36，：	1
20	Early neuronal expression of tumor necrosis factor-quadrature after experimental brain injury contributes to neurological impairment 显示文摘	Knoblach S M Fan L Faden A I	1999	J Neuroimmunol1999，95，：	1

返回顶部每页显示：

共2页首页上一页第1页下一页末页 /2 跳转

互联网出版许可证新出网证(渝)字10号全国400电话 - 免长途话费

用户提示