|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | Protease-activated receptor-2 expression in igA nephropathy: a pbtential role in the pathogenesis of interstitial fibrosis 显示文摘 | Grandaliano G Pontrelli P Cerullo G | 2003 | J Am Soc Nephrol2003，14，： | 1 |
| 2 | CD2AP mu- tations are associated with sporadic nephritic syndrome and focal segmental glomerulosclerosis显示文摘 | Gigante M Pontrelli P Montemumo E | 2009 | Nephrol Dial Trans- plant2009，24，6： | 1 |
| 3 | Type 2 diabetes and bone fragility in children and adults显示文摘Type 2 diabetes(T2D)is a global epidemic disease.The prevalence of T2D in adolescents and young adults is increasing alarmingly.The mechanisms leading to T2D in young people are similar to those in older patients.However,the severity of onset,reduced insulin sensitivity and defective insulin secretion can be different in subjects who develop the disease at a younger age.T2D is associated with different complications,including bone fragility with consequent susceptibility to fractures.The purpose of this systematic review was to describe T2D bone fragility together with all the possible involved pathways.Numerous studies have reported that patients with T2D show preserved,or even increased,bone mineral density compared with controls.This apparent paradox can be explained by the altered bone quality with increased cortical bone porosity and compromised mechanical properties.Furthermore,reduced bone turnover has been described in T2D with reduced markers of bone formation and resorption.These findings prompted different researchers to highlight the mechanisms leading to bone fragility,and numerous critical altered pathways have been identified and studied.In detail,we focused our attention on the role of microvascular disease,advanced glycation end products,the senescence pathway,the Wnt/β-catenin pathway,the osteoprotegerin/receptor-activator of nuclear factor kappa B ligand,osteonectin and fibroblast growth factor 23.The understanding of type 2 myeloid bone fragility is an important issue as it could suggest possible interventions for the prevention of poor bone quality in T2D and/or how to target these pathways when bone disease is clearly evident. | Maria Felicia Faienza Paola Pontrelli Giacomina Brunetti | 2022 | World Journal of Diabetes2022，13，11： | 1 |
| 4 | The differentiation of classickawasa-ki disease,atypical kawasaki disease,and acute adenoviralinfection:Useof clinical features and a rapid direct fluorescent an-tigen test显示文摘 | Barone SR Pontrelli LR Krilov LR | 2000 | ArchPediatrAdolesc Med2000，154，5： | 1 |
| 5 | The pathogenesisof diabetic nephropathy: focus on microRNAs and proteomics显示文摘 | Conserva F Pontrelli P Accetturo M | 2013 | J nephrol2013，26，5： | 1 |
| 6 | Dialysisrelated systemic microinflammation is associated with specific genomic patterns显示文摘 | ZAZA G PONTRELLI P PERTOSA G | 2008 | Nephrol Dial Transplant2008，23，5： | 1 |
| 7 | CD2AP mutations are associated with sporadic nephrotic syndrome and focal segmental glomerulosclerosis (FSGS) 显示文摘 | Gigante M Pontrelli P Montemurno E et ol | 2009 | Nephrol Dial Transplant2009，24，6： | 1 |
| 8 | Calprotectinlevels in meconium显示文摘 | Laforgia N Baldassarre ME Pontrelli G | 2003 | Acta Paediatr2003，92，4： | 1 |
| 9 | CD2AP mutations are associated with sporadic nephrotic syndrome and focal segmental glomerulosclerosis (FSGS) 显示文摘 | Gigante M Pontrelli P Montemurno E | 2009 | Nephrol Dial Transplant2009，24，6： | 1 |
| 10 | CD2AP mutations are associated with sporadic nephrotic syndrome and focal segmental glomerulosclerosis (FSGS)显示文摘 | Gigante M Pontrelli P Montemurno E | 2009 | Nephrol Dial Transplant2009，24，6： | 1 |
| 11 | CD2AP mutations are asso- ciated with sporadic nephrotic syndrome and focal segmental glomerulo- sclerosis(FSGS) 显示文摘 | Gigante M Pontrelli P Montemurno E | 2009 | Nephrol Dial Transplant2009，24，6： | 1 |
| 12 | Laparoscopical managementof cornual pregnancies:a report of three cases显示文摘 | Tinelli A Pellegrino M Pontrelli G | 2010 | European JObstetrics Gynecology and Reproductive Biology2010，151，2： | 1 |
| 13 | CD2AP mutations are associated with sporadic nephrotic syndrome and fo- cal segmental glomerulosclerosis (FSGS) 显示文摘 | GIGANTE M PONTRELLI P MONTEMURNO E | 2009 | Nephrol Dial Trans- plant2009，24，6： | 1 |
| 14 | CD2AP mutations are associated with sporadic nephrotic syndrome and focal segmen- tal glomerulosclerosis (FSGS) 显示文摘 | Gigante M Pontrelli P Montcmurno E | 2009 | Nephrol Dial Transplant2009，24，6： | 1 |
| 15 | The differentiation of classic kawasaki disease, atypical kawasaki disease, and acute adenoviral infection:Use of clinical features and a rapid direct fluorescent antigen test 显示文摘 | Barone SR Pontrelli LR Krilov LR | 2000 | Arch Pediatr Adolesc Med2000，154，5： | 1 |
| 16 | Rapamyein inhibits PAI-1 expression and reduces interstitial fibrosis and glomerulosclerosis in chronic aUograft nephropathy显示文摘 | PONTRELLI P ROSSINI M INFANTE B | 2008 | Transplantation2008，85，1： | 1 |
| 17 | Protease-activated receptor-2 expression in IgA nephropathy:a potential role in the pathogenesis of interstitial fibrosis显示文摘 | Grandaliano G Pontrelli P Cerullo G | 2003 | Am Soc Nephrol2003，14，8： | 1 |
| 18 | Jun-N-terminal kinas regulatesthrombin- induced PAId gene expression in proximal tubular epithelial cells显示文摘 | Pontrelli P Ranier E Ursi M | 2004 | Kidney Int2004，65，6： | 1 |
| 19 | CD2AP mutation are associate with sporadic nephrotic syndrome and focal segmental glomerulosclerosis (FSGS) 显示文摘 | Gigante M Pontrelli P Montemurno E | 2009 | Nephrol Dial Transplant2009，24，6： | 1 |
| 20 | CD40L proinflammatory and profibrotic effects on proximal tubular epithelial cells:role of NF-kappaB and lyn显示文摘 | Pontrelli P Ursi M Ranieri E | 2006 | J Am Soc Nephrol2006，17，3： | 1 |
您所在的机构暂未开通此部分的功能，请联系管理员