Guillain-Barrésyndrome following hepatitis E

查看全文 作　　者：Jean Philippe [1]Loly;Estelle [2]Rikir;Maxime [1]Seivert;Emile [3]Legros;Pierre [4]Defrance;Jacques [1]Belaiche;Gustave [2]Moonen;Jean [1]Delwaide

机构地区：[1]Department of Hepato-Gastroenterology,CHU Sart Tilman,Universitéde Liège;[2]Department of Neurology,CHU Sart Tilman,Universitéde Liège;[3]General Practitioner;[4]Department of Hepato-Gastroenterology,CHPLT La Tourelle

出　　处：《World Journal of Gastroenterology》索引2009年第15卷第13期,共3页

摘　　要：Guillain-Barrésyndrome(GBS)is often triggered by a preceding bacterial or viral infection.Occasionally,it has been observed in association with acute hepatitis A,B and C,and three cases have been previously described in India in which GBS was associated with acute hepatitis E.A molecular mimicry mechanism is supposed to be involved in the pathogenesis of GBS triggered by infectious agents,although the nature of the shared epitopes has not been characterized in most instances,including that in the case of hepatotropic viruses.We report a case of GBS following acute hepatitis E in a European individual.The presence of antiganglioside GM2 antibodies in this patient suggested molecular mimicry involving ganglioside GM2 in the pathogenesis of GBS associated with hepatitis E. 关键词：神经节苷脂戊型肝炎分子模拟病毒性肝炎

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Guillain-Barrésyndrome following hepatitis E

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