K48-1inked ubiquitination and protein degradation regulate 53BP1 recruitment at DNA damage sites

查看全文 作　　者：Frederick A [1,2]Mallette Stephane [1,2]Richard

机构地区：[1]Terry Fox Molecular Oncology Group and the Bloomfield Center for Research on Aging, Sir Mortimer B Davis Jewish GeneralHospital, Segal Cancer Centre, Lady Davis Institute for Medical Research, 3 755 C6te Ste-Catherine Road, Montreal, QuObec, H3T1E2, Canada,;[2]Departments of Medicine and Oncology, McGill University, Montrdal, QuObec, Canada

出　　处：《Cell Research》索引2012年第22卷第8期,共3页

关键词：DNA损伤蛋白降解泛素化 DNA修复肿瘤抑制 DNA断裂蛋白质降解基因突变

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K48-1inked ubiquitination and protein degradation regulate 53BP1 recruitment at DNA damage sites

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