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Apolipoprotein A-I inhibits LPS-induced atherosclerosis in ApoE-/- mice possibly via activated STAT3-mediated upregulation of tristetraprolin

查看全文 作  者:Kai [1,2]YIN;Shi-lin [1,3]TANG;Xiao-hua [1]YU;Guang-hui [4]TU;Rong-fang [5]HE;Jin-feng [1]LI;Di [1,2]XIE;Qing-jun [2]GUI;Yu-chang [6]FU;Zhi-sheng [1]JIANG;Jian [7]TU;Chao-ke [1]TANG 高影响力作者 机构地区:[1]Institute of Cardiovascular Research, Key Laboratory for Atherosclerology of Hunan Province, Life Science Research Center, University of South China, Hengyang 421001, China;[2]Department of Diagnostics, the Medical College, University of South China, Hengyang 421001, China;[3]Department of Intensive Care Unit, the First Affiliated Hospital of University of South China, Hengyang 421001, China;[4]Department of Pathology, Nan Xian Peoples Hospital, Yiyang 413200, China;[5]Department of Pathology, the First Affiliated Hospital, University of South China, Hengyang 421001, China;[6]Department of Nutrition Sciences, University of Alabama at Birmingham, Birmingham, AL 35294-0012, USA;[7]Institute of Pharmacy and Pharmacology, University of South China, Hengyang 421001, China高影响力机构 出  处:《Acta Pharmacologica Sinica》索引2013年第34卷第6期,共10页高影响力期刊 基  金:The authors gratefully acknowledge the financial support from the National Natural Science Foundation of China (81070220, 81170278, and 81100213); the Heng Yang Joint Funds of Hunan Provincial Natural Sciences Foundation of China (10JJ9019); the Hunan Provincial Natural Sciences Foundation of China (06jj5058); the Science & Technology Department Funds of Heng Yang of Hunan Province (2010kj17 and 2010kj41); and the Hunan Provincial Postgraduate Innovation Fund (CX2010B379). 关 键 词:小鼠巨噬细胞 载脂蛋白AI 动脉粥样硬化 STAT3 LPS 载脂蛋白A-I 诱导 激活
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