CD95-CD95L interaction mediates the growth control of MHV68 immortalized B cells by cytotoxic T cells

查看全文 作　　者：Sihan [1,2]Dong;Lingbing [2]Tan;Guifang [1]Chen;Xiaozhen [2]Liang

机构地区：[1]School of Life Science, Shanghai University;[2]Key Laboratory of Molecular Virology & Immunology, Institut Pasteur of Shanghai, Chinese Academy of Sciences

出　　处：《Virologica Sinica》索引2017年第32卷第3期,共3页

基　　金：financially funded by grants from the Natural Science Foundation of China(81371825);the Ministry of Science and Technology of China(2016YFA0502100);the Chinese Academy of Sciences '100 talents' program(2060299) 摘　　要：Dear Editor,The main limitation of the viral pathogenesis study of human gammaherpesviruses Epstein-Barr virus(EBV)and Kaposi sarcoma-associated herpesvirus(KSHV)is the absence of animal model owing to the narrow host tropism of both EBV and KSHV.Murine gammaherpesvirus 68(MHV68)encodes several genes involved 关键词：细胞毒T细胞生长控制 B细胞介导疱疹病毒动物模型发病机制基因编码

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CD95-CD95L interaction mediates the growth control of MHV68 immortalized B cells by cytotoxic T cells

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