Potential role of Müller cells in the pathogenesis of macropsia associated with epiretinal membrane:a hypothesis revisited

查看全文 作　　者：Ahmet [1]Colakoglu;Solmaz Balci [2]Aka

机构地区：[1]Department of Ophthalmology,Acibadem University Schoolof Medicine,Istanbul 34752,Turkey;[2]Department of Ophthalmology,Istanbul University CerrahpasaSchool of Medicine,Istanbul 34098,Turkey

出　　处：《International Journal of Ophthalmology(English edition)》索引2017年第10卷第11期,共9页

摘　　要：Pathophysiological explanations for metamorphopsia associated with retinal pathologies generally focus on photoreceptor organization disruption. However, the retinal microarchitecture is complicated, and we hypothesize that other retinal cells may also be involved. Metamorphopsia has been widely studied in eyes with epiretinal membranes and we revisit the idea that Müller cell displacement causes retinal macropsia. A Pub Med query and related article search for the macula ultrastructure under normal and pathological conditions revealed an enormous amount of information, particularly ultrahigh definition optical coherence tomography and other retinal imaging modality studies. Findings of these imaging studies support our hypothesis that Müller cells, and not cone photoreceptors, are primarily responsible for macropsia in eyes with epiretinal membranes. More specifically, we conclude that displacement of Müller cell endfeet, and not photoreceptor cones, is a more likely the explanation for retinal macropsia associated with epiretinal membranes. 关键词：macropsia metamorphopsia epiretinal membrane Müller cells

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Potential role of Müller cells in the pathogenesis of macropsia associated with epiretinal membrane:a hypothesis revisited

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