NEW INSIGHT INTO MECHANISMS OF AQUEOUS OUTFLOW DEFICIENCY: ROLE OF THE STRESS RESPONSE

查看全文 作　　者：[1]M.E.Fini;[1]K.A.Erickson;[1]E.L.Ginsberg;[1]P.F.Olson;[1]N.Wang;K.R.Wollen-[1]berg;[1]X.Zhang;[1]J.S.Schuman 机构地区：[1]Bascom Palmer Eye Institute, University of Miami, School of Medicine, FL,New England Eye Center, Tufts University, School of Medicine, MA, USA,New England Eye Center, Tufts University, School of Medicine, MA, USA,New England Eye Center, Tufts University, School of Medicine, MA, USA,New England Eye Center, Tufts University, School of Medicine, MA, USA,New England Eye Center, Tufts University, School of Medicine, MA, USA,New England Eye Center, Tufts University, School of Medicine, MA, USA,New England Eye Center, Tufts University, School of Medicine, MA, USA 出　　处：《国际眼科杂志》索引2003年第3卷第1期,共1页 摘　　要：The glaucomas are a group of optic neuropathies comprising the leading cause of irreversible blindness worldwide. Elevated intraocular pressure due to a reduction in normal aqueous outflow is a major causal risk factor. We recently reported that En-dothelial Leukocyte Adhesion Molecule-1 (ELAM-1), the earliest marker for the atherosclerotic plaque in the vasculature, 关 键 词：青光眼 房水外流不足 机制 应激反应 ELAM-1 IL-1