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6篇 您的检索式：作者名="Jessica Fredericks"

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		题名	作者	年代	出处	被引量
	1	Risk Factors Associated with Incident Cataracts and Cataract Surgery in the Age-Related Eye Disease Study (AREDS)显示文摘	Jessica R. Chang Euna Koo Elvira Agrón Joelle Hallak Traci Clemons Dimitri Azar Robert D. Sperduto Frederick L. Ferris Emily Y. Chew	2011	Ophthalmology2011，，11：	1
	2	Electrocardiographic findings in Emergency Department patients with pulmonary embolism显示文摘	Peter B Richman Hassan Loutfi Steven J Lester Patricia Cambell Jessica Matthews Jeremy Friese Joseph Wood David Kasper Frederick Chen Mark Mandell	2004	Journal of Emergency Medicine2004，，2：	1
	3	Human papillomavirus profile of women in Belize City, Belize: correlation with cervical cytopathologic findings显示文摘	Helen P. Cathro Theresa Loya Frederick Dominguez Susan L. Howe Renee Howell Kyle Orndorff Jessica Moreno Elena Mendez Po Chu Fung Natalie L. Beer Peter Allen Alba M. Sosa Kelly K. Gurka Mark H. Stoler Henry F. Frierson		Human Pathology0，，：	1
	4	Does a 48-Hour Rule Predict Outcomes in Patients with Acute Sigmoid Diverticulitis?显示文摘	Jessica Evans Robert Kozol Wayne Frederick Anthony Voytavich William Pennoyer Alexandra Lukianoff Jennifer Lardner	2008	Journal of Gastrointestinal Surgery2008，，7：	1
	5	Ten-Year Incidence Rates of Age-Related Cataract in the Age-Related Eye Disease Study (AREDS): AREDS Report No. 33显示文摘	Euna Koo Jessica R. Chang Elvira Agrón Traci E. Clemons Robert D. Sperduto Frederick L. Ferris Emily Y. Chew	2013	Ophthalmic Epidemiology2013，，2：	1
	6	The role of RAS effectors in BCR/ABL induced chronic myelogenous leukemia显示文摘BCR/ABL is the causative agent of chronic myelogenous leukemia(CML).Through structure/function analysis,several protein motifs have been determined to be important for the development of leukemogenesis.Tyrosine177 of BCR is a Grb2 binding site required for BCR/ABL-induced CML in mice.In the current study,we use a mouse bone marrow transduction/transplantation system to demonstrate that addition of oncogenic NRAS(NRASG12D)to a vector containing a BCR/ABL^(Y177F)mutant“rescues”the CML phenotype rapidly and efficiently.To further narrow down the pathways downstream of RAS that are responsible for this rescue effect,we utilize well-characterized RAS effector loop mutants and determine that the RAL pathway is important for rapid induction of CML.Inhibition of this pathway by a dominant negative RAL is capable of delaying disease progression.Results from the present study support the notion of RAL inhibition as a potential therapy for BCR/ABL-induced CML.	Jessica Fredericks Ruibao Ren	2013	Frontiers of Medicine2013，7，4：	0

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