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19篇 您的检索式:作者名="Orleth"
    题名 作者 年代 出处 被引量
1Targeting tumorassociated endothelial cells: anti- VEGFR2 immunoliposomes mediate tumor vessel disruption and inhibit tumor growth 显示文摘Wicki A Rochlitz C Orleth A 2012Clin Cancer Res2012,18,2:1
2BCR-ABL mediates arsenic trioxide-induced apoptosis independently of its aberrant kinase activity 显示文摘Puccetti E Guller S Orleth A 2000Cancer Res2000,60,13:1
3Targeting tumor-associated endothelial cells:anti-VEGFR2 immunoliposomes mediate tumor vessel disruption and inhibit tumor growth显示文摘Wicki A Rochlitz C Orleth A 2012Clin Cancer Res2012,18,2:1
4Targeting tumor-associated en- dothelial cells: Anti-VEGFR2 immunoliposomes mediate tumor vessel disruption and inhibit tumor growth显示文摘Wicki A Rochlitz C Orleth A 2012Clin Cancer Res2012,18,2:1
5Cell-cycle restriction limits DNA damage and maintains self-renewal of leukaemia stem cells 显示文摘Viale A De F Orleth A 2009Nature2009,457,1:1
6BCR - ABL mediates arsenic trioxide induced apoptosis independently of its aberrant kinase activity 显示文摘Puccett E Guller S Orleth A 2000Cancer Res2000,60,:1
7Bcr-Abl mediates arsenic trioxide-induced apoptosis independently of its aberrant kinase activity显示文摘Puccetti E Guller S Orleth A 2000Cancer Res2000,60,13:1
8BCR-ABL mediates arsenic trioxide induced apoptosis independently of its aberrant kinase activity显示文摘Puccetti E Guller S Orleth A 2000Cancer Res2000,60,13:1
9Cell-cycle restriction limits DNA damage and maintains self-renewal of leukaemia stem cells显示文摘Viale A De Franco F Orleth A 0,,7225:1
10BCR-ABL mediates arsenic trioxide-induced apoptosis independently of its aberrant kinase activity 显示文摘Puccetti E Guller S Orleth A 2000Cancer Res2000,60,:1
11The acute promyelocytic leukaemia specific PML and PLZF proteins localize to adjacent and functionally distinct nuclear bodies显示文摘Ruthardt M Orleth A Tomassoni L 1998Oncogene1998,16,:1
12BCR-ABL mediates arsenic trioxide induced apoptosis independently of its aberrant kinase activity显示文摘Puccett E Guller S Orleth A 2000Cancer Res2000,60,13:1
13Cell - cycle re- striction limits DNA damage and maintains self - renewal of leukaemia stem cells 显示文摘Viale A De Franco F Orleth A 2009Nature2009,457,7225:1
14BCR-ABL mediates arsenic trioxide-induced apoptosis independently of its aberrant kinase activity显示文摘Puccetti E Guller S Orleth A 2000Cancer Res2000,60,13:1
15Cell-cycle restriction limits DNA damage and maintains self-renewal of leukae- mia stem cells 显示文摘Viale A De Franco F Orleth A 2009Nature2009,457,7225:1
16Cell-cycle restriction limits DNA damage and maintains self-renewal of leukaemia stem cells显示文摘Viale A De Franco F Orleth A 2009Nature2009,457,7225:1
17BCR-ABL mediates arsenic trioxide induced apoptosis independently of its aderrant kinaseactivity显示文摘Puccett E Guller S Orleth A et aI 2000Cancer Res2000,60,13:1
18Bcr-abl mediates arsenic trioxide induce apoptosis independently of its aberrant kinase activity 显示文摘Puccetti E Guller S Orleth A 2000Cancer Res2000,60,13:1
19同时靶向VEGFR2和VEGFR3的免疫脂质体显示文摘通过在脂质双层膜中插入抗血管内皮生长因子受体(VEGFR)2和(或)VEGFR3抗体片段,制备了新型抗血管生成的载多柔比星聚乙二醇化脂质体(PLD)。该制品能靶向于表达VEGFR2和VEGFR3的细胞。针对目前临床实践中的靶向制剂都是靶向于VEGFR2的现状。Orleth A 王盈 2015中国医药工业杂志2015,46,8:0
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