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69篇 您的检索式:作者名="Stowe DF"
    题名 作者 年代 出处 被引量
1Mitochondrial reactive oxygen species production in excitable cells:modulators of mitochondrial and cell function显示文摘Stowe DF Camara AK 2009Antioxid Redox Signal2009,11,6:1
2Mitochondrial reactive oxygen species production in excitable cells:modulators of mitochondrial and cell function显示文摘Stowe DF Camara AK 0,,06:1
3Reactive oxygen species as-mediators of cardiac injury and p rotection:the relevance toan-esthesia p ractice显示文摘Kevin LG Novalija E Stowe DF 2005Anesth Analg2005,101,5:1
4Reactive oxygen species as mediators of cardiac injury and protection:the relevance to anesthesia practice显示文摘Kevin LG Novalija E Stowe DF 2005Anesth Analg2005,101,5:1
5Comparison of etomidate, ketamine, midazolam, propofol, and thiopental on function and metabolism of isolated hearts显示文摘Stowe DF Bosnjak ZJ Kampine JP 1992Anesth Analg1992,74,4:1
6Isoflurane modu- lates cardiac mitochondrial bioenergeties by selectively attenuating respiratory complexes 显示文摘Agarwal B Dash RK Stowe DF 2014Biochim Biophys Acta2014,1837,3:1
7Mitochondrial inner membrane electrophysiology assessed by rhodamine-123 transport and fluorescence显示文摘Huang M Camara AK Stowe DF 2007Ann Biomed Eng2007,35,7:1
8Reactive oxygen species as mediators of cardiac injury and protection:The relevance to anesthesia practice显示文摘Kevin LG Novalija E Stowe DF 2005Anesth Analg2005,101,5:1
9Improved contractility and coronary flow in isolated hearts after 1 - day hypothermic preservation with isoflurane is not dependent on K(ATP) channel activation显示文摘Stowe DF Fujita S Bonnjak ZJ 1998Anesth1998,88,1:1
10Ten-hour preservation of guinea pig isolated hearts perfused at low flow with air-saturated Lifor solution at 26{degrees}C:comparison to ViaSpan solution显示文摘Stowe DF Camara AK Heisner JS 2007Am J Physiol Heart Circ Physiol2007,293,1:1
11Mitochondrial reactive oxygenspecies production in excitable cells : modulators of mito-chondrial and cell function 显示文摘Stowe DF Camara AK 2009Antioxid Redox Signal2009,11,6:1
12Modulation of electron transport protects cardiac mitochondria and decreases myocardial injury during ischemia andreperfusion显示文摘Chen Q Camara AK Stowe DF 2007Am J Physiol Cell Physiol2007,292,:1
13Enhanced charge-indep-endent mitochondrial free Ca2+ and attenuated ADP-induced NADH oxidation by isoflurane: implications for cardioprotection 显示文摘Agarwal B Camara AK Stowe DF 2012Biochim Biophys Aeta2012,1817,3:1
14Comparison of etomidate, ketarnine, midazolam, propofol and thiopental on function and metabolism of isolated hearts 显示文摘Stowe DF Bosnjak ZJ Kampine JP 1992Anesth Analg1992,74,4:1
15Cardiac pharmacological preconditioning with volatile anesthetics: from bench to bedside? 显示文摘Riess ML Stowe DF Warltier DC 2004Am J Physiol2004,286,5:1
16Ten-hour preservation of guinea pig isolated hearts perfused at low flow with air-saturated Lifor solution at 26{degrees}C:comparison to ViaSpan solution显示文摘Stowe DF Camara AK Heisner JS 0,,01:1
17Low-flow perfusion of guinea pig isolated hearts with 26 degrees C air-saturated Lifor solution for 20 hours preserves function and metabolism显示文摘Stowe DF Camara AK Heisner JS 0,,09:1
18Reactive oxygen species as mediators of cardiac injury and protection:The relevance to anesthesia practice 显示文摘Kevin LG Novalija E Stowe DF 2005Anesth Analg2005,101,5:1
19Cardiac mitochondrial preconditioning by Big Ca^2+ -sensitive K^+ channel opening requires superoxide radical generation显示文摘Stowe DF Aldakkak M Camara AK 2006Am J Physiol Heart Circ Physiol2006,290,1:1
20Cardiac preconditioning by volatile anesthetic agents:a defining role for altered mitochondrial bioenergetics显示文摘Stowe DF Kevin LG 2004Antioxid Redox Signal2004,6,:1
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