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3篇 您的检索式:作者名="Ye Chengli"
    题名 作者 年代 出处 被引量
1Microstructure and properties of Bi-2223/Ag/Ni tapes显示文摘To reduce the production cost of Bi-2223 superconducting tapes, Bi-2223/Ag/Ni tapes were fabricated by the powder-in-tube process. The effect of heat treatment on the microstructure and critical current of the Bi-2223/Ag/Ni tapes were studied. The phase compositions of the samples were characterized using XRD. The microstructure was observed using SEM. Experimental results indicate that higher temperature is more conducive to the formation of Bi-2223 phase at an atmosphere of 8.5% O 2 . After the two-step heat treatment, the critical current of samples is about 67.5 A.Li Mingya Ye Chengli Liu Jun 2012Rare Metals2012,31,6:2
2Genetic modification of miR-34a enhances efficacy of transplanted human dental pulp stem cells after ischemic stroke显示文摘Human dental pulp stem cells(hDPSCs) promote recovery after ischemic stro ke;however,the therapeutic efficacy is limited by the poor survival of transplanted cells.For in vitro expe riments in the present study,we used oxygen-glucose deprivation/reoxygenation in hDPSCs to mimic cell damage induced by ischemia/reperfusion.We found that miRNA-34a-5p(miR-34a) was elevated under oxygen-glucose deprivation/reoxygenation conditions in hDPSCs.Inhibition of miR-34a facilitated the prolife ration and antioxidant capacity and reduced the apoptosis of hDPSCs.Moreove r,dual-luciferase reporter gene assay showed WNT1and SIRT1 as the targets of miR-34a.In miR-34a knockdown cell lines,WNT1 suppression reduced cell prolife ration,and SIRT1 suppression decreased the antioxidant capacity.Togethe r,these results indicated that miR-34a regulates cell prolife ration and antioxidant stress via targeting WNT1 and SIRT1,respectively.For in vivo expe riments,we injected genetically modified hDPSCs(anti34a-hDPSCs) into the brains of mice.We found that anti34a-hDPSCs significantly inhibited apoptosis,reduced cerebral edema and cerebral infarct volume,and improved motor function in mice.This study provides new insights into the molecular mechanism of the cell prolife ration and antioxidant capacity of hDPSCs,and suggests a potential gene that can be targeted to improve the survival rate and efficacy of transplanted hDPSCs in brain after ischemic stroke.Jianfeng Wang Peibang He Qi Tian Yu Luo Yan He Chengli Liu Pian Gong Yujia Guo Qingsong Ye Mingchang Li 2023Neural Regeneration Research2023,18,9:1
3A dynamic incentive and reputation mechanism for energy-efficient federated learning in 6G显示文摘As 5G becomes commercial,researchers have turned attention toward the Sixth-Generation(6G)network with the vision of connecting intelligence in a green energy-efficient manner.Federated learning triggers an upsurge of green intelligent services such as resources orchestration of communication infrastructures while preserving privacy and increasing communication efficiency.However,designing effective incentives in federated learning is challenging due to the dynamic available clients and the correlation between clients'contributions during the learning process.In this paper,we propose a dynamic incentive and reputation mechanism to improve energy efficiency and training performance of federated learning.The proposed incentive based on the Stackelberg game can timely adjust optimal energy consumption with changes in available clients during federated learning.Meanwhile,clients’contributions in reputation management are formulated based on the cooperative game to capture the correlation between tasks,which satisfies availability,fairness,and additivity.The simulation results show that the proposed scheme can significantly motivate high-performance clients to participate in federated learning and improve the accuracy and energy efficiency of the federated learning model.Ye Zhu Zhiqiang Liu Peng Wang Chenglie Du 2023Digital Communications and Networks2023,9,4:0
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