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152篇 您的检索式:作者名="JAVADOV S"
    题名 作者 年代 出处 被引量
1Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart显示文摘Javadov SA Clarke S Das M 2003J Physio12003,549,:1
2Mitochondrial permeability transition pore opening during myocardial reperfusion-a target for cardioprotection显示文摘Halestrap A P Clarke S J Javadov S A 2004Cardiovasc Res2004,61,3:1
3Mitochondrial permeability transition pore opening during myocardial reperfusion a target for cardioproteetion 显示文摘Halestrap A P Clarke S J Javadov S A 2004Cardiovasc Res2004,61,3:1
4Leptin-induced cardiomyocyte hypertrophy involves selective caveolae and RhoA/ROCK-dependent p38 MAPK translocation to nuclei显示文摘Zeidan A Javadov S Chakrabarti S 0,,01:1
5Mitochondrial permeability transition pore opening during myocardial reperfusion-a target for cardioprotection 显示文摘Halestrap A P Clarke S J Javadov S A 2004Cardiovasc Res2004,61,3:1
6Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart显示文摘HALESTRAP AP KERR PM JAVADOV S 1998Biochim Biophys Acta1998,1366,12:1
7Mitochondrial permeability transition pore opening as a promising therapeutic target in cardiac diseases显示文摘Javadov S Karmazyn M Escobales N 2009J Pharmacol Exp Ther2009,330,3:1
8Mito-chondrial permeability transition pore opening during myocar-dial reperfusion a target for cardioprot ect ion显示文摘HALESTRAP A p CLARKE S J JAVADOV S A 2004Cardiovasc Res2004,61,3:1
9Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injufy of the heart 显示文摘Halestrap AP Kerr PM Javadov S 1998Biochem Biophys Acta1998,1366,12:1
10Crosstalk between mitogen -activated protein kinases and mitochondria in cardiac diseases:therapeutic perspectives 显示文摘Javadov S Jang S Agostini B 2014Pharmacol Ther2014,144,2:1
11Mitochondrial permeability transition and cell death: the role of cyclophilin d显示文摘Javadov S Kuznetsov A 2013Front Physiol2013,4,:1
12Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart显示文摘Javadov SA Clarke S Das M 2003J Physiol2003,549,2:1
13Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection 显示文摘Javadov S Karmazyn M 2007Cell Physiol Biochem2007,20,14:1
14Role of NF-kB andp38 MAPK activation in mediating angiotensin II and endothelin-1 -induced stimulation in leptin production and cardiomyocytehypertrophy显示文摘Rajapurohitam V Kilic A Javadov S 2012Mol Cell Biochem2012,366,12:1
15Essential role of Rho/ROCK-dependent processes and actin dynamics in mediating leptin-induced hypertrophy in rat neonatal ventricular myocytes显示文摘Zeidan A Javadov S Karmazyn M 2006Cardiovasc Res2006,72,1:1
16Eluciting the molecular mechanism of the heart显示文摘Halestrap A P Kerr P M Javadov S 1998Biophys Acta1998,1366,:1
17Mitochondrial permeability transition pore opening as a promising therapeutic target in cardiac diseases显示文摘Javadov S Karmazyn M Escobales N 2009J Pharmacol Exp Ther2009,330,3:1
18Essential role of Rho/ ROCK-dependent processes and actin dynamics in mediating lep-tin-induced hypertrophy in rat neonatal ventricular myocytesJ显示文摘Zeidan A Javadov S Karmazyn M 2006Cardiovasc Res2006,72,1:1
19Mitochondrial permeability transition pore opening during myocardial reperfusion-a target for cardioprofection显示文摘HALESTRAP A P CLARKE S J JAVADOV S A 2004Cardiovasc Res2004,61,3:1
20Mitochondrial permeability transition pore openingasanendpoint toinitiatecell deathandasaputativetarget for cardioprotection显示文摘Javadov S Karmazyn M 2007Cell Physiol Biochem2007,20,14:1
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